Acute Heart Failure by S. M. Ayres (auth.), Dr. Claude Perret, Dr. Jean Louis

By S. M. Ayres (auth.), Dr. Claude Perret, Dr. Jean Louis Vincent (eds.)

This ebook offers a accomplished survey of acute center failure. a chain of specialists famous around the world talk about particular issues in order that, jointly, a large spectrum of experimental and scientific investigations are lined. Pathophysiologic, diagnostic and healing features are integrated. the current quantity may also help clinicians care for the $64000 challenge of acute center failure. it is going to support supply an updated reference for these interested by cardiology, inner medication, pediatrics, anesthesia, extensive care and emergency medicine.

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31. 32. 33. normal volunteers - an assessment by Doppler ultrasonic measurement of ascending aortic blood velocity and acceleration. Am Heart J (in press) Noble MIM, Trenchard D, Guz A (1966) Left ventricular ejection in conscious dogs. Circ Res 19:139-147 Bennett ED, Else W, Miller GAH, Sutton GC, Miller HC, Noble MIM (1974) Maximum acceleration of blood from the left ventricle in patients with ischaemic heart disease. Clin Sci 46:49-59 Mehta N, Bennett ED (1986) Impaired left ventricular function in acute myocardial infarction assessed by Doppler measurements of ascending aortic blood velocity and maximum acceleration.

Kaski that the mechanism by which nitrates are beneficial in patients with dynamic stenosis is not just an effect on myocardial oxygen demand but also an improvement in coronary blood supply [32]. Nitrates can improve myocardial perfusion by dilating constricted collateral vessels, which in some cases might be responsible for the anginal attacks. Altered Vasomotion of Small Vessels This cause of myocardial ischemia appears to be the pathogenetic mechanism in patients with "syndrome X". The diagnosis of this entity is made on the basis of anginal episodes (both exertional and at rest), positive exercise tests, variable effort tolerance, angiographically normal coronary arteries, and no evidence for coronary spasm.

A clinical knowledge of these various factors is necessary for an understanding of the pathophysiological profile of acute cardiac failure and of the hemodynamic effects of any drug with a cardiac and/or a circulatory action. 36 M. C. Aumont et al. References 1. Glantz SA, Parmley WW (1978) Factors which affect the diastolic pressure-volume curve. eire Res 42: 171-180 2. Brutsaert DL, Rademakers FE, Sys SU (1984) Triple control of relaxation: implications in cardiac disease. Circulation 69: 190-196 3.

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